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PRESENTED IN ENGLISH

Biomedical and Health Sciences Conference at the University of Iceland

Breastcancer II

Presenters:

Time, type of presentation:

Session Chair: Sævar Ingþórsson

13:35-13:50: MiR-21-3p influences breast cancer progression
13:50-14:05: Interplay between TGFβ family members and angiogenic factors in breast cancer cells
14:05-14:20: MiR‑99b-5p Suppress the Proliferation and Progression of Triple Negative Breast Cancer Cells
14:20-14:35: GFPT2 is upregulated in breast epithelial–mesenchymal transition and involved in oxidative stress regulation

Abstracts in session A-Z

GFPT2 is upregulated in breast epithelial–mesenchymal transition and involved in oxidative stress regulation

Main author: Qiong Wang. To study metabolic reprogramming in different Epithelial–mesenchymal transition states, we analyzed proteomic changes following EMT in breast gland. We found out enzyme GFPT2 is involved in oxidative stress regulation and a potential target for breast cancer therapy.

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Interplay between TGFβ family members and angiogenic factors in breast cancer cells

Main author: Clara Valls Ferré. This abstract puts in context breast cancer and shows information and recent evidence about disseminated breast cancer cells and their microenvironment, as well as factors (TSP-1 and TGFb) that play an important role in the progression of tumors.

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MiR-21-3p influences breast cancer progression

Arsalan Amirfallah: We analyzed if miR-21-3p expression can predict patient prognosis. Our results indicate that high miR-21-3p expression is associated with a worse prognosis for breast cancer patients and it affects pathways that support breast tumor proliferation.

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MiR‑99b-5p Suppress the Proliferation and Progression of Triple Negative Breast Cancer Cells

Seyedeh Parinaz Mahdavi
This study aimed to determine the expression level of mir-99b-5p in TNBC tissues and study the mir-related cellular parameters. Consequently, the effect of miR-99b-5p on BC cells especially MDA-MB-231 was shown the tumor-suppressor function.

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